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ASA NEWSLETTER
 
 
February 1999
Volume 63
Number 2
   
Does Anesthesia Leave Some of Our Patients With Prolonged Postoperative Cognitive Dysfunction?

Joachim S. Gravenstein, M.D.


We begin the era of the brain. The stage was set last year by two important publications that focused on cognitive dysfunction after anesthesia and surgery in the elderly. A clinical study by Moller and coworkers appeared in The Lancet1 and Dodds and Allison published an excellent review article in the British Journal of Anaesthesia.2

Because cognition may decline with advancing age, Moller et al. studied some 1,200 patients who were 60 years of age and older, assuming that if anesthesia and surgery did indeed affect mental functions, it should be most readily detected in the elderly. The investigators administered psychological tests before the operation, within a week postoperatively and again approximately three months later. In all patients, general anesthesia lasted at least two hours. Cardiac and neurosurgical patients were excluded as were patients who failed to meet the study criteria. Volunteers of comparable age, but not undergoing surgical treatment, were given the same psychological tests at the same intervals to serve as controls.

The investigators detected new cognitive dysfunction in the first postoperative week in some 25 percent of patients. After approximately three months, close to 10 percent of patients showed measurable cognitive dysfunction. This differed significantly from the 3-percent decline in cognitive function observed in the control population over the same time span. The data confirmed the hypothesis that older patients were at greater risk; patients over 70 years of age were twice (14 percent) as likely as those in their 60s (7 percent) to show prolonged cognitive difficulties. The authors rejected the hypotheses that hypoxemia or hypotension during surgery or during the first three postoperative days can be linked to postoperative cognitive dysfunction.

The review article by Dodds and Allison brings this study and many others into focus. The authors cite 89 papers and discuss the history of published concerns about cognitive difficulties after anesthesia. Remarkably, such concerns were expressed with growing frequency only in the last 50 years, perhaps triggered by Bedford's report in 1955 in The Lancet that some elderly patients develop dementia after general anesthesia.3 Dodds and Allison examined publications exploring the mechanisms that might contribute to cognitive dysfunction after anesthesia, among them: drug effects; physiologic changes during anesthesia such as hypoxemia, hypotension and hypocarbia; neurotransmitters; the cholinergic system; and genetic factors. They also reviewed studies that failed to find prolonged postoperative cognitive problems or that compared regional with general anesthesia. Dodds and Allison concluded their review with the following categorical statement: "There is no debate as to whether or not postoperative cognitive deficit exists. It is common and persistent ..."

We may accept as inevitable the frequent psychological disturbances that affect elderly patients in the early postoperative period. Too many mechanisms come to mind: the slow elimination of central nervous system-active drugs used during and after the anesthetic, the effects of metabolic and hormonal disturbances, sleep deprivation, pain and the disorienting effect of the strange hospital environment. The observation, however, that many elderly patients and presumably some younger adults as well show signs of cognitive disturbances three or more months postoperatively present us with new and urgent questions: How many of these disturbances fail to resolve with time? Can such problems push some elderly patients prematurely into dependency? Can we preoperatively identify patients at risk for late postoperative cognitive disturbances? The literature and common sense suggest that patients with pre-existing psychological dysfunction are at higher risk. Can we prevent the problem with the help of different perianesthetic measures? Are the problems even related to anesthesia, or do they have to do with the impact of hospitalization, drugs unrelated to anesthesia, disease processes or changes in living brought about as a consequence of the surgical disease? Whatever the answers to these questions, we should recognize that there is no postoperative complication more frequent and of longer duration than postoperative cognitive dysfunction in the elderly.

Anesthesia is not limited to "putting them to sleep and waking them up again." Over the years, anesthesiologists have assumed responsibilities in the intensive care unit and for pain management for all hospital patients. It is time for the specialty to become actively involved wherever possible in preventing and treating postoperative cognitive dysfunction. Such involvement will call for research and active care of the affected patient.

We may draw a parallel to pain management. While we started out treating only wound pain, we now treat pain of

many different origins. While we started out worrying only about early postanesthetic confusion, we now should recognize that late postoperative cognitive dysfunction demands our attention. At this time, the ASA standards for care and monitoring do not even mention the brain, the very organ we depress so skillfully to relieve anxiety and pain. In the future, it is likely that ASA's standards for care and monitoring will include statements about our responsibilities in preventing postoperative dysfunction of the central nervous system and, where that fails, in monitoring and caring for patients with postoperative dysfunction of the central nervous system.

References:

  1. Moller JT, Cluitmans P, Rasmussen LS, Houx P, et al. Long-term postoperative cognitive dysfunction in the elderly: ISPOCD1 study. ISPOCD investigators. International Study of Post-Operative Cognitive Dysfunction. Lancet. 1998; 351:857-861.
  2. Dodds C, Allison J. Postoperative cognitive dysfunction in the elderly surgical patient. Brit J Anaesth. 1998; 81:449-462.
  3. Bedford PD. Adverse cerebral effects of anaesthesia on old people. Lancet. 1955; 2:259-263.

Joachim S. Gravenstein, M.D., is a Graduate Research Professor Emeritus, Department of Anesthesiology, Shands Teaching Hospital, University of Florida, Gainesville, Florida.


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The views expressed herein are those of the authors and do not necessarily represent or reflect the views, policies or actions of the American Society of Anesthesiologists.

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